Beer potomania
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Phone:617-525-6884
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Overview
This is a syndrome of binge drinking, poor nutritional intake, and profound hyponatremia.
Pathophysiology & Etiology
Hyponatremia is due to a large consumption of beer (which has a poor salt content) together with a minimal intake of ordinary food. A diet poor in salt and protein compounds (i.e. urea precursors) results in reduced excretion of urinary solutes, which limits the ability to excrete free water
References
http://alcalc.oxfordjournals.org/cgi/content/full/35/6/612
Differential Diagnosis
See the differential of hyponatremia
Electrolyte and Biomarker Studies
A value of Uosm < 100 mOsm/kg is indicative of a complete and appropriate suppression of antidiuretic hormone (ADH) secretion, a finding seen with either primary polydipsia (including ‘beer potomania’) or reset osmostat. Criteria used to diagnose the ‘beer potomania’ syndrome include
- A history of binge beer drinking
- Poor dietary intake
- Decreased serum sodium levels
- Absence of other known causes of hyponatremia
Acknowledgements
The content on this page was first contributed by: Editor-In-Chief: C. Michael Gibson, M.S., M.D. [3] Phone:617-525-6884
List of contributors:
Suggested Reading and Key General References
Suggested Links and Web Resources
For Patients
Acknowledgement and Attribution Regarding Sources of Content
Some of the initial content on this page may be incorporated in part from copyleft sources in the public domain including wikis such as Wikipedia and AskDrWiki. Drug information for patients came from the The National Library of Medicine. Infectious disease information may have come from the Centers for Disease Control (CDC). Differential Diagnoses are drawn from clinicians as well as an amalgamation of 3 sources: 1.The Disease Database; 2. Kahan, Scott, Smith, Ellen G. In A Page: Signs and Symptoms. Malden, Massachusetts: Blackwell Publishing, 2004:3; 3. Sailer, Christian, Wasner, Susanne. Differential Diagnosis Pocket. Hermosa Beach, CA: Borm Bruckmeir Publishing LLC, 2002:7 .

