Esophageal web
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Overview
| Esophageal web Classification and external resources | |
| ICD-10 | Q39.4 |
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| DiseasesDB | 31503 |
| eMedicine | med/3413 |
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Most recent articles on Esophageal web Most cited articles on Esophageal web | |
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Ongoing Trials on Esophageal web at Clinical Trials.gov Trial results on Esophageal web Clinical Trials on Esophageal web at Google
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US National Guidelines Clearinghouse on Esophageal web NICE Guidance on Esophageal web
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Patient resources on Esophageal web Discussion groups on Esophageal web Patient Handouts on Esophageal web Directions to Hospitals Treating Esophageal web Risk calculators and risk factors for Esophageal web
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Causes & Risk Factors for Esophageal web | |
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Esophageal webs are thin membranes located in the middle or upper esophagus. They are mainly observed in the Plummer-Vinson syndrome, which is associated with iron deficiency anemia. Its main symptoms are pain and difficulty in swallowing (odynophagia).
Esophageal webs are associated with bullous diseases (such as epidermolysis bullosa, pemphigus, and bullous pemphigoid), with graft versus host disease involving the esophagus, and with celiac disease.
Esophageal webs are thin (2-3mm) membranes of normal esophageal tissue consisting of mucosa and submucosa. They can be congenital or acquired. Congenital webs commonly appear in the middle and inferior third of the esophagus, and they are more likely to be circumferential with a central or eccentric orifice. Acquired webs are much more common than congenital webs and typically appear in the cervical area (postcricoid).
Esophageal webs are more common in white individuals and in women (with a ratio 2:1). The literature describes relations betwean these webs and Plummer-Vinson Syndrome, bullous dermatologic disorders, inlet patch, graft-versus-host disease and celiac disease. The postulated mechanisms are sideropenic anemia (mechanism unknown) or some interference of the immune system. Esophageal webs can be ruptured during upper endoscopy.
Clinical symptoms of this condition are selective (solid more than liquids) dysphagia, thoracic pain, nasopharyngeal reflux, aspiration, perforation and food impaction (the last two are very rare).
Congenital malformations and deformations of digestive system (Q35-Q45, 749-751) | |
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| Tongue, mouth and pharynx | Cleft lip and palate - Van der Woude syndrome - Ankyloglossia - Macroglossia - Pharyngeal pouch |
| Esophagus and upper alimentary tract | Esophageal atresia - Tracheoesophageal fistula - Esophageal web - Pyloric stenosis - Hiatus hernia |
| Intestines | Intestinal atresia (Duodenal atresia) - Imperforate anus - Meckel's diverticulum - Hirschsprung's disease - Intestinal malrotation - Persistent cloaca |
| Pancreas | Annular pancreas - Accessory pancreas - Pancreas divisum |
| Other | Choledochal cysts - Alagille syndrome |
| See also non-congenital (K20-K93, 530-579) | |
Acknowledgement and Attribution Regarding Sources of Content
Some of the initial content on this page may be incorporated in part from copyleft sources in the public domain including wikis such as Wikipedia and AskDrWiki. Drug information for patients came from the The National Library of Medicine. Infectious disease information may have come from the Centers for Disease Control (CDC). Differential Diagnoses are drawn from clinicians as well as an amalgamation of 3 sources: 1.The Disease Database; 2. Kahan, Scott, Smith, Ellen G. In A Page: Signs and Symptoms. Malden, Massachusetts: Blackwell Publishing, 2004:3; 3. Sailer, Christian, Wasner, Susanne. Differential Diagnosis Pocket. Hermosa Beach, CA: Borm Bruckmeir Publishing LLC, 2002:7 .
