Pulseless ventricular tachycardia
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Pulseless Ventricular Tachycardia
In pulseless ventricular tachycardia and electromechanical dissociation, organized electrical activity is present but fails to produce a detectable cardiac output.
In a patient who is in the middle of a cardiac arrest 12 lead electrocardiography is impractical; use a cardiac monitor to determine the rhythm, and any broad complex tachycardia should be assumed to be ventricular in origin.
Pulseless ventricular tachycardia is managed in the same way as ventricular fibrillation, early defibrillation being the mainstay of treatment.[1]
Pulseless Electrical Activity
In pulseless electrical activity the heart continues to work electrically but fails to provide a cardiac output sufficient to produce a palpable pulse.
Electrocardiographic features of pulseless electrical activity
The appearance of the electrocardiogram varies, but several common patterns exist. There may be a normal sinus rhythm or sinus tachycardia, with discernible P waves and QRS complexes. Sometimes there is a bradycardia, with or without P waves, and often with wide QRS complexes.[1]
Successful treatment of pulseless electrical activity depends on whether it is a primary cardiac event or is secondary to a potentially reversible disorder.
Potentially reversible causes of pulseless electrical activity
- Hypovolemia
- Cardiac tamponade
- Tension pneumothorax
- Massive pulmonary embolism
- Hyperkalemia, hypokalemia, and metabolic disorders
- Hypothermia
- Toxic disturbances as overdoses of beta blockers, tricyclic antidepressants, or calcium channel blockers.
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Acknowledgement and Attribution Regarding Sources of Content
Some of the initial content on this page may be incorporated in part from copyleft sources in the public domain including wikis such as Wikipedia and AskDrWiki. Drug information for patients came from the The National Library of Medicine. Infectious disease information may have come from the Centers for Disease Control (CDC). Differential Diagnoses are drawn from clinicians as well as an amalgamation of 3 sources: 1.The Disease Database; 2. Kahan, Scott, Smith, Ellen G. In A Page: Signs and Symptoms. Malden, Massachusetts: Blackwell Publishing, 2004:3; 3. Sailer, Christian, Wasner, Susanne. Differential Diagnosis Pocket. Hermosa Beach, CA: Borm Bruckmeir Publishing LLC, 2002:7 .

